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The pressor response to ketamine hydrochloride injected into intact, unanesthetized canines is accompanied by a tachycardia, rise in cardiac output and a fall in total peripheral resistance. It causes a fall in perfusion strain following a big dose injected into an artificially perfused vascular mattress , and it has little or no potentiating impact uponvasoconstriction responses of epinephrine or norepinephrine. The pressor response to ketamine hydrochloride is lowered or blocked by chlorpromazine (central depressant and peripheral α-adrenergic blockade), by β-adrenergic blockade, and by ganglionic blockade. These observations support the hypothesis that the hypertension produced by ketamine hydrochloride is as a end result of of selective activation of central cardiac stimulating mechanisms leading to an increase in cardiac output. The dog myocardium isn't sensitized to epinephrine and ketamine hydrochloride seems to have a weak antiarrhythmic activity. Well Being Options The m...

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